Phosphoinositide 3-Kinase The fact which the over expression of APP recapitulates many areas of the condition phenotype in mice, and that phenotype is worsened with a selective augmentation of the very most pathogenic A fragment, could very well be the strongest evidence for the causal role of APP and A in sIBM tuskonus October 19, 2024
Phosphoinositide 3-Kinase Moreover, our analysis suggests that DDX11 is involved in anchoring the cohesin complex to the replication machinery during fork progression tuskonus March 2, 2022